Letter Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't Ppublish 15 citations
PubMed: 27418647 DOI: 10.3324/haematol.2016.148106
Genetic inactivation of calpain-1 attenuates pain sensitivity in a humanized mouse model of sickle cell disease.
Haematologica 101(10):e397-e400
Authors: Jennifer O Nwankwo, Jianxun Lei, Jian Xu, Alicia Rivera, Kalpna Gupta, Athar H Chishti
Affiliations
- Graduate Program in Pharmacology and Experimental Therapeutics, Sackler School of Graduate Biomedical Sciences, Tufts University School of Medicine, Boston, MA.
- Vascular Biology Center, Division of Hematology, Oncology and Transplantation, Department of Medicine, University of Minnesota, Minneapolis, MN.
- Children's Medical Center Research Institute, University of Texas Southwestern Medical Center, Dallas, TX.
- Department of Laboratory Medicine, Boston Children's Hospital and Department of Pathology, Harvard Medical School, Boston, MA.
- Graduate Program in Pharmacology and Experimental Therapeutics, Sackler School of Graduate Biomedical Sciences, Tufts University School of Medicine, Boston, MA Department of Developmental, Molecular and Chemical Biology, Tufts University School of Medicine, Boston, MA, USA.
MeSH
Hematology, Anemia, Sickle Cell, Pain, Cells, Calpain
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