Keiki Nagaharu is an expert in Hematology at Mie University (Japan). Keiki Nagaharu has authored 51 publications, the most recent in 2026. SLR score: 160 (community average: 125). How the SLR is computed.

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Keiki Nagaharu Mie University Japan
160 ScienceLeadR Reputation 160
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Hematology

Subspecialties: Oncology - Cardiology - Immunology

Full career – Last 3 years

Main Topics

Publications and Clinical Studies

Publications – Clinical Studies

Profile data updated on

Organizations

Career arc: 2017 - 2026

Source: PubMed

Mie Daigaku Daigakuin Igakukei Kenkyuka Igakubu Ketsueki Shuyo Naikagaku

Publications Synthesis

Sources: PubMed, OpenAlex

Number : 51

Citations

Citations: 251 1st author: 18 Last author: 1 Unique author: 0


Best Journals

Breakdown by type

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Latest Publications

5 most recent indexed articles

Source: Pubmed

Title Journal Year Citations
Chromatin Accessibility Shapes Developmental-Specific Lineage Plasticity in Hematopoiesis.
PMID 42532521 — cells, bias, precursor cell lymphoblastic leukemia-lymphoma, hematopoiesis, microscopy, electron, scanning transmission, chromatin, stem cells, plastics
Blood Adv20260
New insights into JAK2 germline variant-driven stromal cell alterations and proinflammatory bone marrow niche remodeling in myeloproliferative neoplasms.
PMID 41922799 — cells, bone marrow, neoplasms, leukemia, stromal cells
Leukemia20260
VEGF-C-mediated cardiac lymphangiogenesis promotes inflammation resolution in autoimmune acute myocarditis in mice.
PMID 41843753 — cells, genes, immunization, vascular endothelial growth factor a, lymphangiogenesis, fibrosis, heart, macrophages, vascular endothelial growth factor c, cardiovascular system, myocarditis, echocardiography, therapeutics, edema
Cardiovasc Res20260
Embryological cellular origins and hypoxia-mediated mechanisms in PIK3CA-driven refractory vascular malformations.
PMID 40234712 — cells, vascular malformations, vascular endothelial growth factor a, hypoxia, therapeutics
EMBO Mol Med20250
A rare CALR exon 9 mutation p.E379Vfs*53 in AYA generation exhibits gain-of-function nature and can induce essential thrombocythemia; a case report.
PMID 40474352 — thrombocythemia, essential, thrombocytosis, calreticulin, exons, therapeutics, mutation
Hematology20250

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