Osamu Onodera is an expert in Neurology at Niigata University (Japan). As of October 2026, ScienceLeadR ranks Osamu Onodera in the top 1% of experts in Japan for Cerebellar Diseases. Osamu Onodera has authored 509 publications, the most recent in 2026, and is an investigator in 2 clinical trials. SLR score: 1,415 (community average: 125). How the SLR is computed.

LeadR Profile

Osamu Onodera Niigata University Japan
1 415 ScienceLeadR Reputation 1415
Network Map

Neurology

Subspecialties: Genetics - Biochemistry - Public Health

Full career – Last 3 years

Main Topics

Publications and Clinical Studies

Publications – Clinical Studies

Profile data updated on

Organizations

Career arc: 1990 - 2026

Source: PubMed

Niigata University

Publications Synthesis

Sources: PubMed, OpenAlex

Number : 509

Citations

Citations: 14 118 1st author: 13 Last author: 121 Unique author: 9


Breakdown by type

Click a legend label to hide or show it in the chart.

Latest Publications

5 most recent indexed articles

Source: Pubmed

Title Journal Year Citations
Protein-Bound Uremic Toxins and Cognitive Function: A Cross-Sectional Study in a Community-Based Cohort.
PMID 42931513 — indican, cross-sectional studies, cognitive dysfunction, kidney, cognition disorders, cognition
Toxins (Basel)20260
Longitudinal Dynamics of Polyglutamine-Expanded ATXN3 in Biofluids of Spinocerebellar Ataxia Type 3.
PMID 42381263 — spinocerebellar ataxias, multiple chronic conditions, biomarkers, movement disorders, ataxia, plasma, machado-joseph disease
Mov Disord20260
Insertion or deletion variants in TAF15 exon 15 are genetic factors impacting the prognosis of amyotrophic lateral sclerosis in a Japanese cohort.
PMID 42823490 — sclerosis, amyotrophic lateral sclerosis, exons, exome, genetics, medical, mutation, prognosis
Eur J Hum Genet20260
Multicenter Validation of Cerebellar Motor Learning Biomarker for Spinocerebellar Degenerations.
PMID 42573842 — spinocerebellar degenerations, biomarkers, cerebellum, genotype, learning
Cerebellum20260
Persistent export bias of TDP-43 under native autoregulation links insoluble accumulation to nuclear dysfunction.
PMID 42551655 — cells, bias, feedback, 3' untranslated regions, homeostasis, detergents
Neurobiol Dis20260

Clinical Studies 2

Sources: ClinicalTrials, EU CTIS

Synthesis

By phase:

Phase 2 1
Phase 3 1

By status:

Completed 1
Terminated 1

By type:

Interventional 2

By Monocentric/Multicentric:

monocentric 0
multicentric2to4 0
multicentric5to9 0
multicentricup10 2

Main Topics

Cohort/Consortium Investigators 2

LeadR Networks

Main global connection map