Ruben Artero is an expert in Biochemistry at Instituto de Salud Carlos III (Spain). As of August 2026, ScienceLeadR ranks Ruben Artero in the top 1% of experts worldwide for Myotonic Disorders and Muscular Disorders, Atrophic. Ruben Artero has authored 84 publications, the most recent in 2026. SLR score: 322 (community average: 125). How the SLR is computed.

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Biochemistry

Subspecialties: Genetics - Rheumatology - Neurology

Full career – Last 3 years

Main Topics

Publications and Clinical Studies

Publications – Clinical Studies

Profile data updated on

Organizations

Career arc: 1992 - 2026

Source: PubMed

University of Valencia

Publications Synthesis

Sources: PubMed, OpenAlex

Number : 84

Citations

Citations: 4 354 1st author: 3 Last author: 44 Unique author: 0


Best Journals

Breakdown by type

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Latest Publications

5 most recent indexed articles

Source: Pubmed

Title Journal Year Citations
Fatty-acid-based antimiR-23b delivery in the DMSXL model: A potential therapeutic strategy for brain dysfunction in myotonic dystrophy type 1.
PMID 42486096 — oligonucleotides, antisense, myotonic dystrophy, biological science disciplines, rare diseases, brain, alternative splicing, therapeutics, fatty acids
Cell Rep Med20260
The myotonic dystrophy type 1 drug development pipeline: 2026 Edition.
PMID 42208802 — exercise, drug development, oligonucleotides, antisense, myotonic dystrophy, pharmaceutical preparations, drug design, therapeutics, small molecules
Drug Discov Today20260
Oleic Acid Levels in HSA LR Mouse Model of Myotonic Dystrophy Type 1.
PMID 42196193 — muscles, myotonic dystrophy, oleic acid, sex, tissues, fatty acids
Int J Mol Sci20260
Haloperidol induces neuroprotection and enhances neuromuscular function in both murine and human models of spinal muscular atrophy.
PMID 41974892 — muscular atrophy, cells, genes, muscles, spinal cord, microscopy, electron, scanning transmission, atrophy, neuroprotection, haloperidol, therapeutics, muscular atrophy, spinal, spine
Exp Mol Med20260
Enhanced muscle uptake of chemically optimized miR-23b antisense oligonucleotides as lead compounds for myotonic dystrophy type 1.
PMID 41722569 — oligonucleotides, antisense, safety, muscles, myotonic dystrophy, oligonucleotides, micrornas, multiple chronic conditions, myotonia, tissues
Am J Hum Genet20260

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