Thomas Langmann
Thomas Langmann is an expert in Biochemistry at University of Cologne (Germany). As of July 2026, ScienceLeadR ranks Thomas Langmann in the top 1% of experts worldwide for Retinal Diseases. Thomas Langmann has authored 227 publications, the most recent in 2026. SLR score: 724 (community average: 125). How the SLR is computed.
LeadR Profile
Biochemistry
Subspecialties: Ophthalmology - Neurology - Immunology
Main Topics
Publications and Clinical Studies
Profile data updated on
Organizations
Career arc: 1997 - 2026
Number : 227
Citations
Main journals
Best Journals
Breakdown by type
Click a legend label to hide or show it in the chart.
Latest Publications
5 most recent indexed articles
Source: Pubmed
| Title | Journal | Year | Citations |
|---|---|---|---|
| The role of human complement factor H in retinal immune response and the prospects for targeted therapy for age-related macular degeneration (AMD). PMID 42100984 — macular degeneration, complement factor h, immunity, drug delivery systems, complement system proteins, therapeutics, retinaldehyde | Expert Opin Ther Targets | 2026 | 0 |
| Immunomodulation of inflammatory responses preserves retinal integrity in murine models of pericyte-depletion retinopathy. PMID 40591415 — microglia, cells, genes, pericytes, immunomodulation, retina, biomedical research, minocycline, retinaldehyde | JCI Insight | 2025 | 0 |
| Sialylation as a checkpoint for inflammatory and complement-related retinal diseases. PMID 40656681 — microglia, macular degeneration, cells, genes, n-acetylneuraminic acid, retina, phagocytes, complement factor h, immune system, complement system proteins, retinal diseases, retinaldehyde, glycolipids | Front Cell Neurosci | 2025 | 0 |
| Severe Joubert syndrome in family with homozygous POC1B p.Arg106Pro variant is due to a co-inherited deep-intronic mutation in the neighboring CEP290 gene. PMID 40170356 — genes, genetic research, introns, genetic phenomena, mutation | HGG Adv | 2025 | 0 |
| Moss-derived human complement factor H modulates retinal immune response and attenuates retinal degeneration. PMID 40217267 — microglia, cells, genes, retinal degeneration, retina, phagocytes, complement factor h, immunity, tomography, optical coherence, complement system proteins, therapeutics, complement pathway, alternative, retinaldehyde, dna nucleotidylexotransferase | J Neuroinflammation | 2025 | 0 |
LeadR Networks
Main global connection map